Vzkazovník|foxtimes.Self-Assessment Quiz: Plant Dermatitis

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The most common plant rashes may be classified as allergic or chemical irritant dermatitis. Consequently, the allergic contact dermatitis caused by poison ivy requires prior sensitization and occurs many hours after exposure to the plant. Chemical irritant contact dermatitis from plants generally occurs in all individuals exposed to adequate amounts of the chemical.

The threshold for irritation is lowered when the skin barrier is damaged, such as with florists whose hands are constantly wet.

Test your knowledge of these common rashes. True False 2. This is not a true allergy. True False 3. Poison ivy, poison oak and poison sumac are members of the Anacardiaceae family and Toxicodendron genus. True False 4. Members of the family Anacardiaceae do not cause more allergic contact dermatitis than all other plant families combined. True False 5. If the treatment requires systemic steroids, the length of therapy should be 7 days or less.

True False. Poison ivy, or rhus dermatitis, may be prevented if the exposed skin is washed within 15 minutes of exposure. Urushiol, the small molecule that is the allergen in poison ivy, is not water-soluble. Urushiol does not link to receptors on the surface of Langerhans cells found in the epidermis. Irritant contact dermatitis from plants is most often seen in florists.

Persistently wet hands or a prior history of dyshidrotic eczema do not predispose a patient for irritant contact dermatitis. In the rare situation of allergic contact dermatitis from daffodils, the allergen is tulipalin A. Similar black dots found on the skin are not seen on the leaves of the plant. Poison ivy can spread from the blister fluid. Patients with a history of poison ivy react when exposed to cashew nut shell oil, mango fruit skin, crushed berries from the Brazilian pepper tree, or sap from the Japanese lacquer tree.

Exposure to wood smoke from logs covered with poison ivy vines will not cause a severe airborne type of poison ivy in a susceptible person. A severe worsening of poison ivy dermatitis cannot develop from a second contact dermatitis arising from treatment with topical diphenhydramine. Marked scrotal edema is a feature of severe poison ivy. A year-old man, with a past history of poison ivy 50 years ago, can still get poison ivy today. The main goals for treating contact dermatitis are to clear the rash and relieve pruritus.

Treatment options can vary depending on the severity of the reaction. The first step in treatment of all contact dermatitis is to remove the cause of the rash — without doing this, the rash will not resolve. Washing with soap and water, or just water, will remove the poison ivy allergen and other contactants. For irritant contactants, barrier creams, such as Nouriva Repair or Ivy Block, can be a helpful addition to repair the damaged skin barrier. Depending on the severity of the rash, various prescription treatment options are present.

Mild — Mid-potency topical corticosteroids, such as hydrocortisone butyrate 0. A topical anesthetic can be applied to help reduce the pruritus. A combination product of a low-potency topical corticosteroid and an anesthetic, such as pramoxine hydrochloride Enzone, Pramosonemight be the ideal therapy for pediatric patients. Moderate — Mid- to high-potency topical steroids, such as Locoid Lipocream, mometasone furoate Eloconor fluocinonide Lidex, Vanos can be used to relieve the inflammation.

An oral antihistamine can be supplied to help reduce the pruritus and, depending on the selection, help pediatric patients relax at bedtime. Severe — Oral or intramuscular corticosteroids can be provided for rapid relief. Topical application, except for cold-water compresses, is difficult for patients because of the large amount of irritated body surface area.

Ultra-high-potency corticosteroids such as clobetasol propionate Clobetasol, Clobex, Olux and Temovate can be used as well. Part II: Specific features. Cutis Jul; 58 1 : Part I: Prevention —soap and water, topical barriers, hyposensitization. Cutis Jun; 57 6 : J Am Acad Dermatol Aug; 45 2 : Hogan D: Allergic contact dermatitis. Am J Contact Dermat Sep; 8 3 : J Am Acad Dermatol Jan; 4 1 : Guin JD: The black spot test for recognizing poison ivy and related species.

J Am Acad Dermatol Apr; 2 4 : Case 1: Poison ivy, allergic contact dermatitis, rhus dermatitis 1. Sign in. The Dermatologist. About The Dermatologist. Meet Our Editorial Board. Meet Our Editorial Team. Advertising Inquiries. Author Guidelines. Contact Us. Current Issue. Aesthetics Corner. Allergen Focus. Board Review. Clinical Tips. Cover Story. NEA-Reviewed Content. NPF-Approved Content. NRS-Approved Content. The Dermatopathologist. Biological Pathways. Clinical Updates.

Dermatology Advances. FDA Alerts. How I Treat. Sponsored Content. Atopic Dermatitis. Hair and Trichology. Hidradenitis Suppurativa. Infectious Dermatology. Pediatric Dermatology. Practice Advances. Professional Development. Psoriatic Arthritis. Skin Cancer. Skin of Color. Fall Dermatology Week Dermatology Week AAD Annual Meeting.

SDPA Fall. Copied to clipboard. True False Treating Contact Dermatitis from Plants and Flowers The main goals for treating contact dermatitis are to clear the rash and relieve pruritus.

T Case 3: Irritant contact dermatitis 1. F Case 5: Poison ivy 1. T The most common plant rashes may be classified as allergic or chemical irritant dermatitis. Submit Feedback. Email Address. Popular Articles. Results of a Patient Survey on Rosacea Redness.

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Prednisone taper for poison oak emedicine.What is the best duration of steroid therapy for contact dermatitis (rhus)?



    That review suggests starting rhus. Guidelines of care for contact dermatitis. Initially there is pruritis followed by erythema, edema, papules, vesicles, and bullae.

J Am Acad Dermatol Apr; 2 4 : Case 1: Poison ivy, allergic contact dermatitis, rhus dermatitis 1. Sign in. The Dermatologist. About The Dermatologist. Meet Our Editorial Board. Meet Our Editorial Team. Advertising Inquiries. Author Guidelines.

Contact Us. Current Issue. Aesthetics Corner. Allergen Focus. Board Review. Clinical Tips. Cover Story. NEA-Reviewed Content. NPF-Approved Content. NRS-Approved Content. The Dermatopathologist. Biological Pathways. Clinical Updates. Dermatology Advances.

FDA Alerts. How I Treat. Sponsored Content. Atopic Dermatitis. Hair and Trichology. Hidradenitis Suppurativa. Infectious Dermatology. Pediatric Dermatology. Practice Advances. Professional Development. Psoriatic Arthritis. Skin Cancer. Skin of Color. Fall Dermatology Week Dermatology Week AAD Annual Meeting.

SDPA Fall. Copied to clipboard. True False Treating Contact Dermatitis from Plants and Flowers The main goals for treating contact dermatitis are to clear the rash and relieve pruritus.

T Case 3: Irritant contact dermatitis 1. F Case 5: Poison ivy 1. T The most common plant rashes may be classified as allergic or chemical irritant dermatitis. Submit Feedback. That review suggests starting rhus. Brodell RT, Williams L. Taking the itch out of poison ivy: prednisone. Postgrad using prednisone for children with aller- Med ; — Allergic contact dermatitis: Pathophysiology applied to future therapy. Allergic contact dermatitis in Recommendations from others children: A practical approach to management.

Skin Guidelines for treatment of contact Therapy Letter ; —5. Guidelines of care for contact dermatitis. Overview of dermatitis. UpToDate [database].

Stephanides SL. Plant poisoning, toxicodendron. E- removed, or chronic contact dermatitis Medicine [website]. Updated September 25, Available at www. Unless washed off within a few minutes after contact, a reaction will occur. Toxicodendron species contain oleoresins known collectively as urushiol. In susceptible individuals, these compounds trigger a type IV delayed hypersensitivity reaction. Usually, the skin is involved; however, the eyes, airway, and lungs may be involved if exposed to smoke from burning plants.

Reactions from gastrointestinal exposure in the form of urushiol-containing homeopathic remedies have also been reported The greater the sensitivity to the antigen, the sooner and more extensive the reaction. Touching areas of contact can spread the material to other parts of the body that were not initially in contact with the sap and it is important to scrub under the fingernails after contact.

Clinical Manifestations In susceptible individuals, lesions generally appear within hours, although they have been noted to appear earlier. New lesions may continue to appear for up to weeks. Initially, these lesions tend to occur from the slow reaction to adsorbed urushiol; however, lesions that appear later are often secondary to contact with contaminated surfaces.

Initially there is pruritis followed by erythema, edema, papules, vesicles, and bullae. Helpful in diagnosing the rash is the linear distribution caused by the branches brushing the area of contact. Scratching the rash will help spread the lesions and there may be areas of rash where the skin was protected by clothing. Contrary to popular belief, the fluid from vesicles and bulla do not spread the rash.

The rash can be spread from contact with fomites like shoes, clothing, tools, and from the smoke from burning plants.

Animal fur can also be reservoirs of the sap and pets can help spread the antigen if they have been in contact with the plants. Complete resolution is expected within days. Treatment Immediate decontamination. Urushiol penetrates the skin and binds to membrane lipids within minutes of contact. If the toxin can be removed before this occurs, reaction can be avoided.

Washing exposed areas with copious amounts of water within 20 minutes of exposure has been shown to reduce reactivity.

Scant evidence exists for the best duration of steroid therapy for contact dermatitis due to plants rhus. Review articles recommend 10 to 21 days of treatment with topical or oral corticosteroids for moderate to severe contact dermatitis due to plants strength of recommendation [SOR]: Cbased on review articles.

The primary reason given for the duration of 2 to 3 weeks is to prevent rebound dermatitis. Evidence for the best treatment of rhus dermatitis is negligible. Most recommendations stem from review articles and expert opinion. Rhus dermatitis is one example of a disorder for which we must fall back on our logic and personal experience.

Since the painful itchy blisters and erythema from the oleoresin may take up to 1 week to appear, and because the rash may persist for more than 2 weeks, it makes sense to prescribe oral steroids in severe cases for longer than the usual 5- to 7-day burst. Habif, a popular dermatology text, suggests gradually tapering steroids from 60 to 10 mg over a day course.

No published studies compare varying durations of treatment with steroids for contact dermatitis due to plants, including rhus.

Many review articles refer to rebound dermatitis when using courses of oral steroids such as Medrol dosepaks for fewer than 14 days. One case report noted failure of a tapering dose over 5 days of oral methylprednisolone for treatment of poison ivy contact dermatitis. The systemic treatments listed include oral or intramuscular corticosteroids, but no discussion of duration is mentioned. UpToDate discusses avoidance of the offending substance for 2 to 4 weeks, use of topical corticosteroids of medium to strong potency for a limited time without defining the durationand use of systemic corticosteroids in severe cases, prescribing a course of prednisone at 40 mg daily for 4 to 6 days followed by 20 mg for 4 to 6 days.

Because the rash may persist for more than 2 weeks, it makes sense to prescribe oral steroids for longer than 5 or 7 days. Skip to main content. Clinical Inquiries. What is the best duration of steroid therapy for contact dermatitis rhus? J Fam Pract. Meadows, MLS. PDF Download. Evidence-based answers from the Family Physicians Inquiries Network.

The oral corticosteroid is tapered over a 2-week period, but a complicated tapering regimen probably is not necessary given the short duration. eMedicine states that although oral systemic steroids, with a taper of prednisone over 10 to 14 days, are the standard for severe toxicodendron dermatitis, some. Scant evidence exists for the best duration of steroid therapy for Brodell RT, Williams L. Taking the itch out of poison ivy: prednisone.4 A third. Topical steroids are useful for mild cases. May need oral steroids for weeks. Full dosage for the first week and then taper the second week to prevent. Most review articles recommend systemic steroids for severe poison ivy con- tact dermatitis, but these articles do not define “severe,” describe the taper. Poison oak is most common west of the Rockies, poison ivy to the east, and poison Sumac in the southeast. Skin Cancer. Are patients with skin of color Coding for Repairs and Excisions.

To browse Academia. Scant evidence exists for the best duration of steroid therapy for contact dermatitis due to plants rhus. Review articles recommend 10 to 21 days of treatment with topical or oral corticosteroids for moderate to severe contact dermatitis due to plants strength of recommendation [SOR]: C, based on review articles.

The primary reason given for the duration of 2 to 3 weeks is to prevent rebound dermatitis. Log in with Facebook Log in with Google. Remember me on this computer. Enter the email address you signed up with and we'll email you a reset link. Need an account?

Click here to sign up. Download Free PDF. What is the best duration of steroid therapy for contact dermatitis rhus? Kevin Craig. Abstract Scant evidence exists for the best duration of steroid therapy for contact dermatitis due to plants rhus. The primary reason rhus. Review articles recommend 10 to 21 days given for the duration of 2 to 3 weeks is to of treatment with topical or oral corticosteroids prevent rebound dermatitis.

Habif, a popular dermatology text, Evidence for the best treatment of rhus dermatitis suggests gradually tapering steroids from 60 to is negligible. Most recommendations stem from 10 mg over a day course. Rhus dermatitis randomized controlled trials and remembering my is one example of a disorder for which we must patient who bounced back after I only gave 1 week fall back on our logic and personal experience. That review suggests starting rhus. Brodell RT, Williams L.

Taking the itch out of poison ivy: prednisone. Postgrad using prednisone for children with aller- Med ; — Allergic contact dermatitis: Pathophysiology applied to future therapy. Allergic contact dermatitis in Recommendations from others children: A practical approach to management.

Skin Guidelines for treatment of contact Therapy Letter ; —5. Guidelines of care for contact dermatitis. Overview of dermatitis. UpToDate [database].

Stephanides SL. Plant poisoning, toxicodendron. E- removed, or chronic contact dermatitis Medicine [website]. Updated September 25, Available at www. Taylor JS. Contact dermatitis and related disorders. Updated September, Available at: moderate to severe cases. The systemic www. Accessed on January 10, Habif TP. Clinical Dermatology. Louis, Mo: Mosby; Failure of a tapering dose of oral methylprednisolone to treat reactions to poison ivy. JAMA ; Time Factors Glucocorticoids Topical steroid.



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